Reviewed by medical experts • For informational purposes only
G6PD deficiency types are grouped by enzyme activity into WHO classes I to V, with common variants and carrier states explained.
G6PD deficiency has many gene variants. They differ in how low the enzyme level is and how often problems occur. Doctors often describe them in simplified classes.
WHO classes (simplified)
Common named variants
The exact variant is found only by gene testing, which is not usually needed for care.
By clinical picture
Carriers and females
Females may be carriers with mild or no symptoms, or partially or fully deficient.
Ayurvedic naming
Ayurveda has no equivalent. Pallor and yellowness are discussed under pandu and kamala, but these are traditional, interpretive categories and not the same condition.
Why the type matters
A more severe variant means stricter avoidance of triggers. Ask your doctor what your result means for you.
Written By
Dr. Daksh
BDS
Reviewed By
Dr. Varun Gupta
MD Pharmacology, MBBS
Last updated on
27 Sep 2022 • 06:23 PM (IST)
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Inherited X-linked enzyme gene variants cause G6PD deficiency; triggers such as fava beans, some drugs and infections set off red cell breakdown.
G6PD deficiency does not progress in stages; this page explains baseline, trigger, haemolysis and recovery phases, and testing timing.
Preventing G6PD crises means avoiding triggers, newborn and family screening, telling every doctor, and treating infections early.
Some medicines and household products can trigger G6PD haemolysis. Which to avoid, safer choices, and why to ask a doctor first.
G6PD deficiency diet: avoid fava beans, eat balanced meals, check labels and supplements, and know no food or herb cures the condition.
G6PD deficiency cannot be cured; care focuses on trigger avoidance, treating haemolysis, transfusion if needed, and newborn jaundice care.
